高盐/肾素-血管紧张素-醛固酮系统与骨代谢
High salt/renin-angiotensin-aldosterone system and bone metabolism
  
DOI:10.3969/j.issn.1006.7108.2017.11.024
中文关键词:  高盐  钠离子;肾素-血管紧张素-醛固酮系统  骨代谢  骨丢失;骨质疏松
英文关键词:High salt  Sodium  RAAS  Bone metabolism  Bone loss  Osteoporosis
基金项目:国家自然科学基金青年基金项目(81400853);广东 省普通高校国际暨港澳台合作创新平台及国际合作重大项目 (2015KGJHZ022);中央财政支持地方高校发展专项资金(粵财政 [2016]202 号)
作者单位
陈瑞晗1,2 陈珺1,2* 1.广东药科大学生命科学与生物制药学院,广东 广州510006 2.广东药科大学生物资源与创新药物研究中心,广东 广州510006 
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中文摘要:
      近年来,越来越多的研究证实高盐摄人除了导致高血压,还会影响骨代谢过程。而机体的水盐代谢主要受肾素-血管紧张素-醛固酮系统调节,亦有研究发现该系统具有参与调节骨代谢的作用。本文综述近年来对高盐/肾素-血管紧张素-醛固酮系统影响骨代谢及其机制的相关研究,旨在揭示水盐代谢与骨代谢相关性的理论依据。
英文摘要:
      In recent years,more and more studies confirm that high salt intake may not only lead to hypertensive,but also affect bone metabolism. Renin-angiotensin-aldosterone system ( RAAS) plays an important role in regulation of water and salt metabolism,as well as in regulation of bone metabolism. This review focuses on the effects of high salt-intake and RAAS on bone metabolism and its mechanism, in order to provide theoretical basis for the correlation between water and salt metabolism and bone metabolism.
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