外泌体circRNAs ITCH/miRNAs靶向调控成骨分化、骨生成机制
Study on the mechanism of exosome circRNAs ITCH/miRNAs targeted regulation of osteogenic differentiation and osteogenesis
  
DOI:10.3969/j.issn.1006-7108.2025.05.010
中文关键词:  外泌体  circRNAs ITCH  miR-23a-5p  骨髓间充质干细胞  卵巢切除大鼠
英文关键词:exosomes  circRNAs ITCH  miR-23a-5p  bone marrow mesenchymal stem cells  ovariectomized rats
基金项目:湖南省卫生健康委科研计划项目(202202256321)
作者单位
徐娣 魏伟强* 李莎 李文姝 蒋腾龙 长沙市第四医院(湖南师范大学附属长沙医院)骨一科湖南 长沙 410006 
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中文摘要:
      目的 探讨外泌体(exosomes,Exo)来源环状RNA(circular RNAs,circRNAs)ITCH在骨髓间充质干细胞(bone marrow mesenchymal stem cells,BMSCs)成骨分化中的调控作用及分子机制。方法 将来自卵巢切除大鼠(OVX-Exo)和假手术大鼠(Sham-Exo)的BMSCs来源外泌体与骨髓来源的巨噬细胞共培养,以研究它们对破骨细胞分化的影响。为了阐明ITCH对BMSCs的成骨分化影响,将BMSCs分为Sham-Exo(假手术)组、OVX-Exo(卵巢切除)组、OVX-Exo+mimic-NC组和OVX-Exo+ITCH mimic组。为了证实ITCH通过抑制miR-23a-5p表达促进成骨细胞增殖和分化潜能,将BMSCs分为OVX-Exo+ITCH+mimic-NC组和OVX-Exo+ITCH+miR-23a-5p mimic组。通过定量实时PCR (qRT-PCR)确定BMSCs中ITCH、miR-23a-5p表达。分别通过CCK-8、茜素红染色和ALP染色检测细胞增殖、钙化结节和细胞活性情况。结果 与Sham-Exo组相比,OVX-Exo组TRAP阳性细胞的数量和破骨细胞标记物的mRNA表达显著升高(P<0.05)。OVX-Exo中ITCH mRNA表达水平显著低于Sham-Exo(P < 0.01)。在BMSCs的成骨分化期间,ITCH表达在成骨诱导后的第5、7、9天显著增加(P < 0.01)。与Sham-Exo组相比,OVX-Exo组和OVX-Exo+mimic-NC组BMSCs细胞增殖显著降低(P < 0.05),而OVX-Exo+ITCH mimic组BMSCs细胞增殖显著高于OVX-Exo+mimic-NC组(P < 0.05)。与Sham-Exo组相比,OVX-Exo组和OVX-Exo+mimic-NC组BMSCs细胞ALP活性和矿物质沉积显著降低(P < 0.05),而OVX-Exo+ITCH mimic组BMSCs细胞ALP活性和矿物质沉积显著高于OVX-Exo+mimic-NC组(P < 0.05)。miR-23a-5p mimic显著降低了ITCH-WT报告载体的荧光素酶活性,和miR-23a-5p抑制剂显著增加了ITCH-WT报告载体的荧光素酶活性。与OVX-Exo+ITCH mimic+mimic-NC相比,OVX-Exo+ITCH mimic+miR-23a-5p mimic组BMSCs细胞增殖、ALP活性和矿物质沉积均显著降低(P < 0.05)。结论 ITCH在介导Exo调节骨形成和骨吸收中发挥重要作用,其过表达通过海绵miR-23a-5p促进BMSCs的成骨分化。
英文摘要:
      Objective To investigate the regulatory role and molecular mechanism of extracellular vesicle (Exo) derived circular RNA (circRNAs) ITCH in osteogenic differentiation of bone marrow mesenchymal stem cells (BMSCs). Methods Exosomes derived from BMSCs from ovariectomized rats (OVX-Exo) and sham-operated rats (Sham-Exo) were co-cultured with macrophages derived from bone marrow to study their effects on osteoclast differentiation. In order to clarify the effect of ITCH on osteogenic differentiation of BMSCs, BMSCs were divided into Sham-Exo group, OVX-Exo group, OVX-Exo+mimic-NC group and OVX-Exo+ITCH mimic group. In order to confirm the potential of ITC to promote osteoblast proliferation and differentiation by inhibiting the expression of miR-23a-5p, BMSCs were divided into OVX-Exo+ITCH+mimic-NC group and OVX-Exo+ITCH+miR-23a-5p mimic group. The expression of ITCH and miR-23a-5p in BMSCs was determined by quantitative real-time PCR (qRT-PCR). Cell proliferation, calcified nodules and cell activity were detected by CCK-8, alizarin red staining and ALP staining respectively. Results Compared with the Sham-Exo group, the number of TRAP positive cells and the mRNA expression of osteoclast markers in OVX-Exo group increased significantly (P<0.05). The expression level of ITCH mRNA in OVX-Exo was lower than that in Sham-Exo(P< 0.01). During the osteogenic differentiation of BMSCs, the expression of ITCH increased significantly on the 5th, 7th and 9th day after osteogenic induction (P<0.01). Compared with Sham-Exo group, the proliferation of BMSCs in OVX-Exo group and OVX-Exo+mimic-NC group decreased significantly (P<0.05), while the proliferation of BMSCs in OVX-Exo+ITCH mimic group was higher than that in OVX-Exo+mimic-NC group (P<0.05). The ALP activity and mineral deposition of BMSCs in OVX-Exo group and OVX-Exo+mimic-NC group were lower than those in Sham-Exo group (P< 0.05), while the ALP activity and mineral deposition of BMSCs in OVX-Exo+ITCH mimic group were significantly higher than those in OVX-Exo+mimic-NC group (P<0.05). MiR-23a-5p mimic significantly reduced the luciferase activity of ITC-WT reporter vector, and miR-23a-5p inhibitor significantly increased the luciferase activity of ITC-WT reporter vector. Compared with OVX-Exo+ITCH mimic+mimic-NC, the proliferation, ALP activity and mineral deposition of BMSCs in OVX-EXO+Itchmimic+Mir-23a-5p Mimic group decreased significantly (P<0.05). Conclusion ITCH plays an important role in mediating Exo to regulate bone formation and bone resorption, and its overexpression promotes osteogenic differentiation of BMSCs through sponge miR-23a-5p.
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