由“脑-肠-骨轴”探讨左归丸缓解SOP小鼠骨量丢失及海马退变的作用机制
The mechanism of the relief of bone mass loss and hippocampal degeneration with Zuogui Pill based on the brain-gut-bone axis in SOP mice
  
DOI:10.3969/j.issn.1006-7108.2026.01.004
中文关键词:  左归丸  老年性骨质疏松  肠道菌群  脑-肠-骨轴  海马退变  骨量丢失  OPG/RANK/RANKL通路
英文关键词:Zuogui pill  senile osteoporosis  intestinal flora  brain-gut-bone axis  hippocampal degeneration  loss of bone mass  OPG/RANK/RANKL pathway
基金项目:山西省重点研发计划项目(201703D421031)
作者单位
吴倩瑶1 雒荣荣2 张亚江2 王艳2 许凯霞3 王颖莉4* 1.山西中医药大学护理学院山西 晋中 030619 2.山西中医药大学中药与食品工程学院山西 晋中 030619 3.山西中医药大学基础医学学院山西 晋中 030619 4.山西中医药大学实验管理中心山西 晋中 030619 
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中文摘要:
      目的 基于“脑-肠-骨轴”初步探讨左归丸对老年性骨质疏松症(senile osteoporosis,SOP)模型小鼠海马区神经元退行性病变、肠道菌群变化及骨量丢失的作用。方法 连续12周腹腔注射D-半乳糖(120mg/kg)构建SOP模型,将小鼠随机分为空白组、模型组、左归丸高剂量组、左归丸低剂量组;予以左归丸药物干预8周,随后进行Morris水迷宫实验检测小鼠认知功能。取材后检测各组脑组织氧化应激指标,尼氏染色法检测海马尼式神经元完整性,16S rRNA检测肠道菌群多样性,小动物X光机检测小鼠骨量丢失情况,小鼠血清检测骨代谢指标,免疫组织化学法检测骨代谢通路相关蛋白,采用spearman分析法对血清骨代谢因子、脑组织氧化应激因子与肠道菌群中的差异菌群进行关联分析。结果与Model组相比,左归丸给药组可显著缩短逃避潜伏期时间(P<0.05);显著增加海马区尼式小体数量(P<0.01);提高脑组织抗氧化酶含量(P<0.05);在门水平上,Model组小鼠肠道菌群中Bacteroidota相对丰度显著增高(P<0.01),左归丸给药组Verrucomicrobiota等菌群相对丰度显著升高(P<0.05),Bacteroidota相对丰度显著下降(P<0.05)。小鼠股骨干骺端与骨干区的骨量丢失得到改善,显著改善血清骨代谢指标(P<0.01),显著提高OPG/RANKL蛋白含量比值(P<0.01)。相关性分析显示,过氧化氢酶与肠道菌群中的Proteobacteria呈正相关(P<0.01),与Prevotellaceae_NK3B31_group呈负相关(P<0.01)。结论左归丸依据“脑-肠-骨轴”改善SOP小鼠肠道菌群丰度,进而缓解海马神经元退行性病变,调节OPG/RANK/RANKL通路缓解骨量丢失。
英文摘要:
      Objective Based on the brain-gut-bone axis, the effects of Zuogui Pill on neuronal degeneration in the hippocampus, changes in intestinal flora, and bone loss were investigated in senile mice osteoporosis (SOP) model. Methods The SOP model was constructed with intraperitoneal injection of D-galactose (120 mg/kg) for 12 consecutive weeks. The mice were randomly divided into the blank group, the model group, the Zuogui Pill high-dose group, and the Zuogui Pill low-dose group. Mice received Zuogui Pill for 8 weeks, and then the Morris water maze experiment was carried out to detect the cognitive function of the mice. Brain tissue oxidative stress indexes were detected in each group after sampling. Hippocampal nidus neuron integrity was detected with Nissen staining. Intestinal flora diversity was detected using 16S rRNA. Bone loss was detected with small animal X-ray machine. Bone metabolism indexes were detected in mice serum. Bone metabolism pathway-related proteins were detected with immunohistochemistry. The results of the analysis of serum bone metabolism factors, brain tissue oxidative stress factors, and the cognitive function of mice in the intestinal flora were investigated using Spearman's assay. The association analysis of serum bone metabolism factor, brain tissue oxidative stress factor, and differential flora in intestinal flora was carried out with Spearman analysis. Results Compared with the model group, the Zuogui Pill administration group significantly shortened the escape latency time (P<0.05), significantly increased the number of nidamental vesicles in the hippocampus (P<0.01), and increased the content of antioxidant enzymes in the brain tissue (P<0.05). The relative abundance of Bacteroidota in the intestinal flora of the model group mice increased significantly at the gate level (P<0.01). The relative abundance of Verrucomicrobiota and other bacterial flora was significantly higher (P<0.05) and the relative abundance of Bacteroidota was significantly lower (P<0.05) in the Zuogui Pill administration group. Bone loss in the femoral epiphysis and diaphysis region of mice was ameliorated. Serum bone metabolism indexes improved significantly (P<0.01). The ratio of OPG/RANKL protein content increased significantly (P<0.01). Correlation analysis showed that catalase was positively correlated with Proteobacteria in intestinal flora (P<0.01) and negatively correlated with Prevotellaceae in NK3B31 group (P<0.01). Conclusion Zuogui Pill improves the abundance of intestinal flora in SOP mice according to the brain-gut-bone axis, which in turn alleviates the degenerative neuronal lesions in the hippocampus, and regulates the OPG/RANK/RANKL pathway to alleviate the loss of bone mass.
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