淫羊藿次苷Ⅱ治疗骨质疏松的基础理论研究进展
Research progress in the basic theory of icariside II in the treatment of osteoporosis
  
DOI:10.3969/j.issn.1006-7108.2026.01.016
中文关键词:  淫羊藿次苷Ⅱ  骨质疏松  生物利用度  外泌体  信号通路
英文关键词:icariside II  osteoporosis  bioavailability  exosomes  signaling pathways
基金项目:国家自然科学基金(82374164)
作者单位
张东顺 李东* 南京中医药大学附属医院江苏 南京 210004 
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中文摘要:
      骨质疏松症(osteoporosis,OP)是一种以骨量减少、骨微结构退化及骨脆性增加为特征的全身性代谢性骨病。其发生机制涉及成骨与破骨过程的动态失衡。淫羊藿次苷Ⅱ(IcarisideⅡ,ICSⅡ)是淫羊藿的主要活性成分,因其多靶点、多层次的作用机制,在骨质疏松治疗中展现了良好潜力。该文总结了近5年关于ICSⅡ抗骨质疏松的研究进展,探讨其通过MAPK/ERK、Wnt/β-catenin、EGFR-Akt-Nrf2等信号通路调控成骨分化、抑制破骨细胞活性及改善骨代谢平衡的药理作用。此外,针对ICSⅡ水溶性差、生物利用度低的问题,综述了通过纳米技术、磷脂复合物及外泌体载药等策略提高其生物利用度的研究进展。尽管ICS Ⅱ已在体内外研究中展现出一定效果,但其长期安全性及临床转化仍需进一步探索。
英文摘要:
      Osteoporosis (OP) is a systemic metabolic bone disease characterized by decreased bone mass, degradation of bone microarchitecture, and increased bone fragility. Its pathogenesis involves a dynamic imbalance between osteogenesis and bone resorption. Icariside II (ICS II), the main active ingredient of Epimedium, has shown significant potential in the treatment of OP due to its multi-target and multi-level mechanisms of action. This paper reviews recent advancements in ICS II research since the past five years, focusing on its pharmacological effects, including regulating osteogenic differentiation via MAPK/ERK, Wnt/β-catenin, and EGFR-Akt-Nrf2 signaling pathways, inhibiting osteoclast activity, and improving bone metabolic balance. Furthermore, this review discusses strategies to enhance the bioavailability of ICS II, such as nanotechnology, phospholipid complexes, and exosome-based drug delivery systems, addressing challenges associated with its poor solubility and low bioavailability. Despite promising findings from in vitro and in vivo studies, further exploration is needed to assess the long-term safety and clinical applicability of ICS II.
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