Piezo1在肌少-骨质疏松症中作用机制研究进展
Advances in the mechanism of Piezo1 in sarcopenia and osteoporosis
  
DOI:10.3969/j.issn.1006-7108.2026.01.018
中文关键词:  机械敏感性离子通道蛋白Piezo1  肌少-骨质疏松症  钙离子  肌骨稳态  骨骼-肌肉轴
英文关键词:mechanosensitive ion channel protein Piezo1  sarcopenia-osteoporosis  calcium ion  musculoskeletal homeostasis  skeleton-muscle axis
基金项目:甘肃省联合科研基金(24JRRA902);甘肃省中医院横向课题(20180401);兰州市科技计划项目(2023-2-20,2023-ZD-49)
作者单位
孙兴翔1 孔令俊2* 宋承鑫1 邓叶龙2 李想1 张金磊1 韩升龙2 秦浩1 1.甘肃中医药大学甘肃 兰州730000 2.甘肃省中医院甘肃 兰州730050 
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中文摘要:
      Piezo1是一种机械敏感离子通道蛋白,在机械刺激转化为生物信号传导中具有关键作用。可响应力学刺激、与细胞骨架相互作用、介导Ca2+内流激活相关胞内信号通路;影响细胞增殖、分化等多种生物学过程。肌肉骨骼系统负责维持结构支撑、运动和体内平衡。近年来研究发现,Piezo1在肌骨代谢疾病中扮演重要角色,可维持“肌骨稳态”,并调控肌少-骨质疏松症进程。本文对Piezo1在肌骨衰减疾病中作用机制进行总结,探究其通过调控相关信号通路促进骨髓间充质干细胞成骨分化,抑制破骨生成,维持机体骨稳态;促进成肌细胞分化与肌管形成,维持卫星细胞功能,帮助肌肉修复及再生,改善肌肉萎缩的作用机制。总结与展望目前研究中存在的问题,以期为调控“肌骨稳态”提供新方向。
英文摘要:
      Piezo1 is a mechanosensitive ion channel protein that plays a key role in the conversion of mechanical stimuli into biological signaling. It can respond to mechanical stimulation, interact with cytoskeleton, and mediate Ca2+ influx to activate related intracellular signaling pathways. It affects many biological processes such as cell proliferation and differentiation. The musculoskeletal system is responsible for maintaining structural support, movement and homeostasis. In recent years, Piezo1 has been found to play an important role in musculoskeletal metabolic diseases, maintaining "musculoskeletal homeostasis" and regulating the process of sarcopenia and osteoporosis. In this paper, the mechanism of Piezo1 in musculoskeletal decay diseases was summarized, and it was explored that PIEZO1 promoted osteogenic differentiation of bone marrow mesenchymal stem cells, inhibited osteoclast formation, and maintained bone homeostasis by regulating related signaling pathways. Promote myoblast differentiation and muscle tube formation, maintain satellite cell function, help muscle repair and regeneration, and improve the mechanism of muscle atrophy. In order to provide a new direction for the regulation of musculoskeletal homeostasis, the existing problems in current research are summarized and prospeced.
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